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Its ability to cross the blood–brain barrier in laboratory models, combined with a relatively long plasma half-life compared to other small neuropeptides, has contributed to its enduring relevance in neurochemistry research

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One class includes the clinically important etoposide, doxorubicin, and mitoxantrone, and these drugs “convert” topoisomerase II into a DNA-damaging agent by increasing the levels of topoisomerase II/DNA covalent complexes and DNA double-stranded breaks (DSBs)